Is that what you think it said? Are you that dumb? This is too complex for a simplistic thinker like you but here you go
The study shows that genes play a small and limited role in determining sexuality. Genetic heritability — all of the information stored in our genes and passed between generations — can only explain 8 to 25 percent of why people have same-sex relations, based on the study’s results.
Moreover, the researchers found that sexuality is
polygenic — meaning hundreds or even thousands of genes make tiny contributions to the trait. That pattern is similar to other heritable (but complex) characteristics like height or a
proclivity toward trying new things. (Things like red/green colorblindness, freckles and dimples
can be traced back to single genes). But polygenic traits can be strongly influenced by the environment, meaning there’s no clear winner in this “nature versus nurture” debate.
Let me know when you have completed your study that contradicts what this study found.
“Obviously, there are environmental causes of sexual orientation. We knew that before this study.” said Bailey,
citing the well-defined role that life experiences play in sexual development. “But that doesn’t mean we know how to manipulate sexual orientation mentally.”
What the scientists did
The study set out to investigate a 20-year-old genetics debate in sexuality by combing through two huge collections of DNA profiles: the UK Biobank and 23andMe.
With
a reported 9 million users in its database, 23andMe is arguably the most popular, direct-to-consumer DNA testing company on the planet.
This GWAS study found that, like with many human behaviors, sexuality doesn’t have a strong genetic backing.
When the team looked for DNA patterns that had strong correlations, they found that no one gene could account for any more than 1 percent of people’s sexuality. The strongest signals came from five random genes.
Two of those genes correlated with same-sex sexuality in males, one of which is known to influence the sense of smell. One gene cropped up for females and two others showed solid patterns in both males and females. But their individual scores never passed this 1-percent mark — meaning they are all minor contributors to same-sex sexual behavior.
When the team looked more broadly across all the genomes — across the thousands of genes that they screened for the nearly 500,000 subjects — the genes similarities they found could only account for 8 to 25 percent of same-sex sexual behavior.
“It’s effectively impossible to predict an individual’s sexual behavior from their genome,” said Ben Neale, a geneticist at Massachusetts General Hospital and the Broad Institute who led the study. “Genetics is less than half of this story for sexual behavior.”
Humans have tried to understand human sexuality for centuries — and genetics researchers joined the fray in the early 1990s after a series of studies on twins suggested homosexuality ran in families. These kinds of studies have continued through the years, going as far as pinpointing a gene on the X chromosome — Xq28 — as the culprit.
“As a teenager trying to understand myself and understand my sexuality, I looked at the internet for “the gay gene” and obviously came across Xq28,” said Fah Sathirapongsasuti, a study co-author and senior scientist at 23andMe, which he joked once led him to believe he inherited his gayness from his mother.
His comments speak to the larger narrative about using biology to define complex behaviors — like sexuality — when science is always evolving and takes time to find anything close to definitive.
They did attempt to examine some elements of this continuum by conducting GWAS analysis on three smaller DNA databases wherein the participants had been surveyed using the Kinsey Scale. The Kinsey Scale is a somewhat infamous test for determining the strength of a person’s feelings toward members of the same- and opposite-sexes. In other words, it tries to judge if a person leans gay, straight or bisexual.
The team found genetics cannot explain people’s scores on the Kinsey Scale.
“We discovered that the Kinsey Scale … is really an oversimplification of the diversity of sexual behavior in humans,” Neale said. Bailey disagrees, arguing that people’s feelings of sexual interest and arousal — and therefore, their readouts on the Kinsey Scale — may be too complicated to validate through genetics.
He did agree with Neale that the debate is now closed on whether any single gene is responsible for sexual orientation.
“[Our study] underscores an important role for the environment in shaping human sexual behavior and perhaps most importantly there is no single gay gene but rather the contribution of many small genetic effects scattered across the genome,” Neale said.