Where is this mountain of evidence for evolution?

[PredFan]

I am not an atheist, but I'm a believer in evolution. I also understand that things change in real science and nothing in true science is ever settled. Evidence and data may from time to time change the theory of evolution but that doesn't make it any less true.

I must respectfully point out to you that what you accuse ashes lists of doing is exactly what Creationists ALWAYS do. They never attempt to prove Creationism, they only attack evolution.

Why should we have to defend something that is so obvious? It takes a special kind of stupid to be ignorant of the obvious fact that the universe and everything in it, is the result of an intelligent Creator.

To believe that everything created itself from nothing, without any help at all, is sheer lunacy.

The problem with you is an identical problem throughout the population who believes in Creationism; you don't know the first thing about evolution and you form your opinions based on ignorance. Here you are confusing the Big Bang Theory with Evolution.

I have confused nothing. the Big Bang, Abiogenesis and evolution are all part of the same thing. A naturalistic origin of life. I may not be an expert on evolution, but I will debate you on it anytime you like. you say I don't understand it. I assure you, that I do. so, you want to debate it? bring it on. I'll even let you choose the topic.

Well you can't debate it if you don't know shit about it and it's obvious that you don't know shit about it.

So, you're backing down? You're refusing to debate me? You do realize how that makes you look, don't you? You've just confirmed my suspician. You're a blowhard and a coward. Otherwise, you'd jump at the opportunity to make look like a fool.

I can't debate someone who has no knowledge of the subject, if you want to label it backing down just to save your ignorant pride, do whatever you want.

 

[Book of Jeremiah]

No one can refute anything you say. Invincible ignorance is called invincible ignorance because it is invincible.

Eternal ignorance is what happens when you refuse to examine the facts that prove your "theories" are wrong.

 

[Book of Jeremiah]

so what new species did your bacteria become ?

Which one? There are many.

When has any Antibiotic resistant microorganisms been observed becoming another species

Antibiotic resistance

Antibiotic resistance is the ability of a microorganism to withstand the effects of an antibiotic.

It is a specific type of drug resistance.

Antibiotic resistance evolves naturally via natural selection through random mutation, but it could also be engineered by applying an evolutionary stress on a population.

Once such a gene is generated, bacteria can then transfer the genetic information in a horizontal fashion (between individuals) by plasmid exchange.

If a bacterium carries several resistance genes, it is called multiresistant or, informally, a superbug.

Causes Antibiotic resistance can also be introduced artificially into a microorganism through transformation protocols.

This can be a useful way of implanting artificial genes into the microorganism.

Antibiotic resistance is a consequence of evolution via natural selection.

The antibiotic action is an environmental pressure; those bacteria which have a mutation allowing them to survive will live on to reproduce.

They will then pass this trait to their offspring, which will be a fully resistant generation.

Several studies have demonstrated that patterns of antibiotic usage greatly affect the number of resistant organisms which develop.

Overuse of broad-spectrum antibiotics, such as second- and third-generation cephalosporins, greatly hastens the development of methicillin resistance.

Other factors contributing towards resistance include incorrect diagnosis, unnecessary prescriptions, improper use of antibiotics by patients, and the use of antibiotics as livestock food additives for growth promotion.

Researchers have recently demonstrated the bacterial protein LexA may play a key role in the acquisition of bacterial mutations.

Resistant pathogens Staphylococcus aureus (colloquially known as "Staph aureus" or a Staph infection) is one of the major resistant pathogens.

Found on the mucous membranes and the skin of around a third of the population, it is extremely adaptable to antibiotic pressure.

It was the first bacterium in which penicillin resistance was found—in 1947, just four years after the drug started being mass-produced.

Methicillin was then the antibiotic of choice, but has since been replaced by oxacillin due to significant kidney toxicity.

MRSA (methicillin-resistant Staphylococcus aureus) was first detected in Britain in 1961 and is now "quite common" in hospitals.

MRSA was responsible for 37% of fatal cases of blood poisoning in the UK in 1999, up from 4% in 1991.

Half of all S. aureus infections in the US are resistant to penicillin, methicillin, tetracycline and erythromycin.

This left vancomycin as the only effective agent available at the time.

However, strains with intermediate (4-8 ug/ml) levels of resistence, termed GISA (glycopeptide intermediate Staphylococcus aureus) or VISA (vancomycin intermediate Staphylococcus aureus), began appearing the the late 1990s.

The first identified case was in Japan in 1996, and strains have since been found in hospitals in England, France and the US.

The first documented strain with complete (>16ug/ml) resistence to vancomycin, termed VRSA (Vancomycin-resistant Staphylococcus aureus) appeared in the United States in 2002.

A new class of antibiotics, oxazolidinones, became available in the 1990s, and the first commercially available oxazolidinone, linezolid, is comparable to vancomycin in effectiveness against MRSA.

Linezolid-resistance in Staphylococcus aureus was reported in 2003.

CA-MRSA (Community-acquired MRSA) has now emerged as an epidemic that is responsible for rapidly progressive, fatal diseases including necrotizing pneumonia, severe sepsis and necrotizing fasciitis.

Methicillin-resistant Staphylococcus aureus (MRSA) is the most frequently identified antimicrobial drug-resistant pathogen in US hospitals.

The epidemiology of infections caused by MRSA is rapidly changing.

In the past 10 years, infections caused by this organism have emerged in the community.

The 2 MRSA clones in the United States most closely associated with community outbreaks, USA400 (MW2 strain, ST1 lineage) and USA300, often contain Panton-Valentine leukocidin (PVL) genes and, more frequently, have been associated with skin and soft tissue infections.

Outbreaks of community-associated (CA)-MRSA infections have been reported in correctional facilities, among athletic teams, among military recruits, in newborn nurseries, and among active homosexual men.

CA-MRSA infections now appear to be endemic in many urban regions and cause most CA-S. aureus infections.

Enterococcus faecium is another superbug found in hospitals.

Penicillin-Resistant Enterococcus was seen in 1983, Vancomycin-Resistant Enterococcus (VRE) in 1987, and Linezolid-Resistant Enterococcus (LRE) in the late 1990s.

Streptococcus pyogenes (Group A Streptococcus: GAS) infections can usually be treated with many different antibiotics.

Early treatment may reduce the risk of death from invasive group A streptococcal disease.

However, even the best medical care does not prevent death in every case.

For those with very severe illness, supportive care in an intensive care unit may be needed.

For persons with necrotizing fasciitis, surgery often is needed to remove damaged tissue.

Strains of S. pyogenes resistant to macrolide antibiotics have emerged, however all strains remain uniformly sensitive to penicillin.

Resistance of Streptococcus pneumoniae to penicillin and other beta-lactams is increasing worldwide.

The major mechanism of resistance involves the introduction of mutations in genes encoding penicillin-binding proteins.

Selective pressure is thought to play an important role, and use of beta-lactam antibiotics has been implicated as a risk factor for infection and colonization.

Streptococcus pneumoniae is responsible for pneumonia, bacteremia, otitis media, meningitis, sinusitis, peritonitis and arthritis.

so the short answer is never..the bacteria remains a bacteria

Brilliant. But irrelevant.

Hardly. Eots is right on the mark here but your pride simply refuses to admit the possibility that you are wrong.

 

[Book of Jeremiah]

Why should we have to defend something that is so obvious? It takes a special kind of stupid to be ignorant of the obvious fact that the universe and everything in it, is the result of an intelligent Creator.

To believe that everything created itself from nothing, without any help at all, is sheer lunacy.

The problem with you is an identical problem throughout the population who believes in Creationism; you don't know the first thing about evolution and you form your opinions based on ignorance. Here you are confusing the Big Bang Theory with Evolution.

I have confused nothing. the Big Bang, Abiogenesis and evolution are all part of the same thing. A naturalistic origin of life. I may not be an expert on evolution, but I will debate you on it anytime you like. you say I don't understand it. I assure you, that I do. so, you want to debate it? bring it on. I'll even let you choose the topic.

Well you can't debate it if you don't know shit about it and it's obvious that you don't know shit about it.

So, you're backing down? You're refusing to debate me? You do realize how that makes you look, don't you? You've just confirmed my suspician. You're a blowhard and a coward. Otherwise, you'd jump at the opportunity to make look like a fool.

I can't debate someone who has no knowledge of the subject, if you want to label it backing down just to save your ignorant pride, do whatever you want.

The only ignorance and pride I'm seeing here is yours, Predfan. Your I can't debate excuse is nothing but a cop out. The truth is you have no answers to refute the evidence that proves evolution to be a lie.

 

[orogenicman]

Which one? There are many.

When has any Antibiotic resistant microorganisms been observed becoming another species

Antibiotic resistance

Antibiotic resistance is the ability of a microorganism to withstand the effects of an antibiotic.

It is a specific type of drug resistance.

Antibiotic resistance evolves naturally via natural selection through random mutation, but it could also be engineered by applying an evolutionary stress on a population.

Once such a gene is generated, bacteria can then transfer the genetic information in a horizontal fashion (between individuals) by plasmid exchange.

If a bacterium carries several resistance genes, it is called multiresistant or, informally, a superbug.

Causes Antibiotic resistance can also be introduced artificially into a microorganism through transformation protocols.

This can be a useful way of implanting artificial genes into the microorganism.

Antibiotic resistance is a consequence of evolution via natural selection.

The antibiotic action is an environmental pressure; those bacteria which have a mutation allowing them to survive will live on to reproduce.

They will then pass this trait to their offspring, which will be a fully resistant generation.

Several studies have demonstrated that patterns of antibiotic usage greatly affect the number of resistant organisms which develop.

Overuse of broad-spectrum antibiotics, such as second- and third-generation cephalosporins, greatly hastens the development of methicillin resistance.

Other factors contributing towards resistance include incorrect diagnosis, unnecessary prescriptions, improper use of antibiotics by patients, and the use of antibiotics as livestock food additives for growth promotion.

Researchers have recently demonstrated the bacterial protein LexA may play a key role in the acquisition of bacterial mutations.

Resistant pathogens Staphylococcus aureus (colloquially known as "Staph aureus" or a Staph infection) is one of the major resistant pathogens.

Found on the mucous membranes and the skin of around a third of the population, it is extremely adaptable to antibiotic pressure.

It was the first bacterium in which penicillin resistance was found—in 1947, just four years after the drug started being mass-produced.

Methicillin was then the antibiotic of choice, but has since been replaced by oxacillin due to significant kidney toxicity.

MRSA (methicillin-resistant Staphylococcus aureus) was first detected in Britain in 1961 and is now "quite common" in hospitals.

MRSA was responsible for 37% of fatal cases of blood poisoning in the UK in 1999, up from 4% in 1991.

Half of all S. aureus infections in the US are resistant to penicillin, methicillin, tetracycline and erythromycin.

This left vancomycin as the only effective agent available at the time.

However, strains with intermediate (4-8 ug/ml) levels of resistence, termed GISA (glycopeptide intermediate Staphylococcus aureus) or VISA (vancomycin intermediate Staphylococcus aureus), began appearing the the late 1990s.

The first identified case was in Japan in 1996, and strains have since been found in hospitals in England, France and the US.

The first documented strain with complete (>16ug/ml) resistence to vancomycin, termed VRSA (Vancomycin-resistant Staphylococcus aureus) appeared in the United States in 2002.

A new class of antibiotics, oxazolidinones, became available in the 1990s, and the first commercially available oxazolidinone, linezolid, is comparable to vancomycin in effectiveness against MRSA.

Linezolid-resistance in Staphylococcus aureus was reported in 2003.

CA-MRSA (Community-acquired MRSA) has now emerged as an epidemic that is responsible for rapidly progressive, fatal diseases including necrotizing pneumonia, severe sepsis and necrotizing fasciitis.

Methicillin-resistant Staphylococcus aureus (MRSA) is the most frequently identified antimicrobial drug-resistant pathogen in US hospitals.

The epidemiology of infections caused by MRSA is rapidly changing.

In the past 10 years, infections caused by this organism have emerged in the community.

The 2 MRSA clones in the United States most closely associated with community outbreaks, USA400 (MW2 strain, ST1 lineage) and USA300, often contain Panton-Valentine leukocidin (PVL) genes and, more frequently, have been associated with skin and soft tissue infections.

Outbreaks of community-associated (CA)-MRSA infections have been reported in correctional facilities, among athletic teams, among military recruits, in newborn nurseries, and among active homosexual men.

CA-MRSA infections now appear to be endemic in many urban regions and cause most CA-S. aureus infections.

Enterococcus faecium is another superbug found in hospitals.

Penicillin-Resistant Enterococcus was seen in 1983, Vancomycin-Resistant Enterococcus (VRE) in 1987, and Linezolid-Resistant Enterococcus (LRE) in the late 1990s.

Streptococcus pyogenes (Group A Streptococcus: GAS) infections can usually be treated with many different antibiotics.

Early treatment may reduce the risk of death from invasive group A streptococcal disease.

However, even the best medical care does not prevent death in every case.

For those with very severe illness, supportive care in an intensive care unit may be needed.

For persons with necrotizing fasciitis, surgery often is needed to remove damaged tissue.

Strains of S. pyogenes resistant to macrolide antibiotics have emerged, however all strains remain uniformly sensitive to penicillin.

Resistance of Streptococcus pneumoniae to penicillin and other beta-lactams is increasing worldwide.

The major mechanism of resistance involves the introduction of mutations in genes encoding penicillin-binding proteins.

Selective pressure is thought to play an important role, and use of beta-lactam antibiotics has been implicated as a risk factor for infection and colonization.

Streptococcus pneumoniae is responsible for pneumonia, bacteremia, otitis media, meningitis, sinusitis, peritonitis and arthritis.

so the short answer is never..the bacteria remains a bacteria

Brilliant. But irrelevant.

Hardly. Eots is right on the mark here but your pride simply refuses to admit the possibility that you are wrong.

On the mark? The principles of evolution have allowed us to not only discover the agents of disease, but have allowed us to discover antibiotics, and how the resistance to them works so we can develop better treatments. You don't have to believe it. You can just thank all those tireless lab workers who are trying to make your life better. So no he is not only not on the mark, he is flat out wrong.

 

[orogenicman]

The problem with you is an identical problem throughout the population who believes in Creationism; you don't know the first thing about evolution and you form your opinions based on ignorance. Here you are confusing the Big Bang Theory with Evolution.

I have confused nothing. the Big Bang, Abiogenesis and evolution are all part of the same thing. A naturalistic origin of life. I may not be an expert on evolution, but I will debate you on it anytime you like. you say I don't understand it. I assure you, that I do. so, you want to debate it? bring it on. I'll even let you choose the topic.

Well you can't debate it if you don't know shit about it and it's obvious that you don't know shit about it.

So, you're backing down? You're refusing to debate me? You do realize how that makes you look, don't you? You've just confirmed my suspician. You're a blowhard and a coward. Otherwise, you'd jump at the opportunity to make look like a fool.

I can't debate someone who has no knowledge of the subject, if you want to label it backing down just to save your ignorant pride, do whatever you want.

The only ignorance and pride I'm seeing here is yours, Predfan. Your I can't debate excuse is nothing but a cop out. The truth is you have no answers to refute the evidence that proves evolution to be a lie.

What evidence, where? No one has posted any evidence here to refute evolution. If you have some, by all means, have it published in a peer reviewed science journal so the rest of us can review it and have some input it the matter.

 

[eots]

When has any Antibiotic resistant microorganisms been observed becoming another species

Antibiotic resistance

Antibiotic resistance is the ability of a microorganism to withstand the effects of an antibiotic.

It is a specific type of drug resistance.

Antibiotic resistance evolves naturally via natural selection through random mutation, but it could also be engineered by applying an evolutionary stress on a population.

Once such a gene is generated, bacteria can then transfer the genetic information in a horizontal fashion (between individuals) by plasmid exchange.

If a bacterium carries several resistance genes, it is called multiresistant or, informally, a superbug.

Causes Antibiotic resistance can also be introduced artificially into a microorganism through transformation protocols.

This can be a useful way of implanting artificial genes into the microorganism.

Antibiotic resistance is a consequence of evolution via natural selection.

The antibiotic action is an environmental pressure; those bacteria which have a mutation allowing them to survive will live on to reproduce.

They will then pass this trait to their offspring, which will be a fully resistant generation.

Several studies have demonstrated that patterns of antibiotic usage greatly affect the number of resistant organisms which develop.

Overuse of broad-spectrum antibiotics, such as second- and third-generation cephalosporins, greatly hastens the development of methicillin resistance.

Other factors contributing towards resistance include incorrect diagnosis, unnecessary prescriptions, improper use of antibiotics by patients, and the use of antibiotics as livestock food additives for growth promotion.

Researchers have recently demonstrated the bacterial protein LexA may play a key role in the acquisition of bacterial mutations.

Resistant pathogens Staphylococcus aureus (colloquially known as "Staph aureus" or a Staph infection) is one of the major resistant pathogens.

Found on the mucous membranes and the skin of around a third of the population, it is extremely adaptable to antibiotic pressure.

It was the first bacterium in which penicillin resistance was found—in 1947, just four years after the drug started being mass-produced.

Methicillin was then the antibiotic of choice, but has since been replaced by oxacillin due to significant kidney toxicity.

MRSA (methicillin-resistant Staphylococcus aureus) was first detected in Britain in 1961 and is now "quite common" in hospitals.

MRSA was responsible for 37% of fatal cases of blood poisoning in the UK in 1999, up from 4% in 1991.

Half of all S. aureus infections in the US are resistant to penicillin, methicillin, tetracycline and erythromycin.

This left vancomycin as the only effective agent available at the time.

However, strains with intermediate (4-8 ug/ml) levels of resistence, termed GISA (glycopeptide intermediate Staphylococcus aureus) or VISA (vancomycin intermediate Staphylococcus aureus), began appearing the the late 1990s.

The first identified case was in Japan in 1996, and strains have since been found in hospitals in England, France and the US.

The first documented strain with complete (>16ug/ml) resistence to vancomycin, termed VRSA (Vancomycin-resistant Staphylococcus aureus) appeared in the United States in 2002.

A new class of antibiotics, oxazolidinones, became available in the 1990s, and the first commercially available oxazolidinone, linezolid, is comparable to vancomycin in effectiveness against MRSA.

Linezolid-resistance in Staphylococcus aureus was reported in 2003.

CA-MRSA (Community-acquired MRSA) has now emerged as an epidemic that is responsible for rapidly progressive, fatal diseases including necrotizing pneumonia, severe sepsis and necrotizing fasciitis.

Methicillin-resistant Staphylococcus aureus (MRSA) is the most frequently identified antimicrobial drug-resistant pathogen in US hospitals.

The epidemiology of infections caused by MRSA is rapidly changing.

In the past 10 years, infections caused by this organism have emerged in the community.

The 2 MRSA clones in the United States most closely associated with community outbreaks, USA400 (MW2 strain, ST1 lineage) and USA300, often contain Panton-Valentine leukocidin (PVL) genes and, more frequently, have been associated with skin and soft tissue infections.

Outbreaks of community-associated (CA)-MRSA infections have been reported in correctional facilities, among athletic teams, among military recruits, in newborn nurseries, and among active homosexual men.

CA-MRSA infections now appear to be endemic in many urban regions and cause most CA-S. aureus infections.

Enterococcus faecium is another superbug found in hospitals.

Penicillin-Resistant Enterococcus was seen in 1983, Vancomycin-Resistant Enterococcus (VRE) in 1987, and Linezolid-Resistant Enterococcus (LRE) in the late 1990s.

Streptococcus pyogenes (Group A Streptococcus: GAS) infections can usually be treated with many different antibiotics.

Early treatment may reduce the risk of death from invasive group A streptococcal disease.

However, even the best medical care does not prevent death in every case.

For those with very severe illness, supportive care in an intensive care unit may be needed.

For persons with necrotizing fasciitis, surgery often is needed to remove damaged tissue.

Strains of S. pyogenes resistant to macrolide antibiotics have emerged, however all strains remain uniformly sensitive to penicillin.

Resistance of Streptococcus pneumoniae to penicillin and other beta-lactams is increasing worldwide.

The major mechanism of resistance involves the introduction of mutations in genes encoding penicillin-binding proteins.

Selective pressure is thought to play an important role, and use of beta-lactam antibiotics has been implicated as a risk factor for infection and colonization.

Streptococcus pneumoniae is responsible for pneumonia, bacteremia, otitis media, meningitis, sinusitis, peritonitis and arthritis.

so the short answer is never..the bacteria remains a bacteria

Brilliant. But irrelevant.

Hardly. Eots is right on the mark here but your pride simply refuses to admit the possibility that you are wrong.

On the mark? The principles of evolution have allowed us to not only discover the agents of disease, but have allowed us to discover antibiotics, and how the resistance to them works so we can develop better treatments. You don't have to believe it. You can just thank all those tireless lab workers who are trying to make your life better. So no he is not only not on the mark, he is flat out wrong.

That was not the work of ..The principles of evolution

 

[Book of Jeremiah]

No, he is not wrong. And neither was Dr. Kent Hovind wrong which was proven when he defeated the evolution promoters at university after university. Perhaps that is why the Govt. trumped up charges against him and put him in prison for the past 99 months and counting - for depositing 9600 dollars into his own bank account. Utterly ridiculous. You people cannot debate the facts and win so you put those who present them best behind bars. What weakness!

 

[Book of Jeremiah]

Dr. Hovind proves the truth about evolution. It's a lie.:

 

[Hollie]

No, he is not wrong. And neither was Dr. Kent Hovind wrong which was proven when he defeated the evolution promoters at university after university. Perhaps that is why the Govt. trumped up charges against him and put him in prison for the past 99 months and counting - for depositing 9600 dollars into his own bank account. Utterly ridiculous. You people cannot debate the facts and win so you put those who present them best behind bars. What weakness!

Kent Hovind is a laughable joke.

Encyclopedia of American Loons Search results for hovind

 

[eots]

No, he is not wrong. And neither was Dr. Kent Hovind wrong which was proven when he defeated the evolution promoters at university after university. Perhaps that is why the Govt. trumped up charges against him and put him in prison for the past 99 months and counting - for depositing 9600 dollars into his own bank account. Utterly ridiculous. You people cannot debate the facts and win so you put those who present them best behind bars. What weakness!

Kent Hovind is a laughable joke.

Encyclopedia of American Loons Search results for hovind

Your fave link is joke

 

[eots]

funny how real science sounds like science and evolution sounds like philosophy...

 

[Hollie]

No, he is not wrong. And neither was Dr. Kent Hovind wrong which was proven when he defeated the evolution promoters at university after university. Perhaps that is why the Govt. trumped up charges against him and put him in prison for the past 99 months and counting - for depositing 9600 dollars into his own bank account. Utterly ridiculous. You people cannot debate the facts and win so you put those who present them best behind bars. What weakness!

Kent Hovind is a laughable joke.

Encyclopedia of American Loons Search results for hovind

Your fave link is joke

Its not at all surprising that you would defend a charlatan and a hack such as Hovind. Not surprisingly, you couldn't refute a single element of the article.

 

[Hollie]

funny how real science sounds like science and evolution sounds like philosophy...

That's due to your appalling ignorance of the subject matter.

 

[Book of Jeremiah]

funny how real science sounds like science and evolution sounds like philosophy...

 

[eots]

the only other science so contested is man made climate change which seems also to be plagued with hidden agenda and a mixture of science.,politics and philosophy

 

[Uncensored2008]

No worries, batshit crazy wackos, here you go, five pieces of 'factual' evidence.

In this article, we look at five simple examples which support the Theory of Evolution.

by Richard Peacock

Five Proofs of Evolution Evolution FAQ

1. The universal genetic code. All cells on Earth, from our white blood cells, to simple bacteria, to cells in the leaves of trees, are capable of reading any piece of DNA from any life form on Earth. This is very strong evidence for a common ancestor from which all life descended.





2. The fossil record. The fossil record shows that the simplest fossils will be found in the oldest rocks, and it can also show a smooth and gradual transition from one form of life to another.


Please watch this video for an excellent demonstration of fossils transitioning from simple life to complex vertebrates.





3. Genetic commonalities. Human beings have approximately 96% of genes in common with chimpanzees, about 90% of genes in common with cats (source), 80% with cows (source), 75% with mice (source), and so on. This does not prove that we evolved from chimpanzees or cats, though, only that we shared a common ancestor in the past. And the amount of difference between our genomes corresponds to how long ago our genetic lines diverged.





4. Common traits in embryos. Humans, dogs, snakes, fish, monkeys, eels (and many more life forms) are all considered "chordates" because we belong to the phylum Chordata. One of the features of this phylum is that, as embryos, all these life forms have gill slits, tails, and specific anatomical structures involving the spine. For humans (and other non-fish) the gill slits reform into the bones of the ear and jaw at a later stage in development. But, initially, all chordate embryos strongly resemble each other.


In fact, pig embryos are often dissected in biology classes because of how similar they look to human embryos. These common characteristics could only be possible if all members of the phylum Chordata descended from a common ancestor.





5. Bacterial resistance to antibiotics. Bacteria colonies can only build up a resistance to antibiotics through evolution. It is important to note that in every colony of bacteria, there are a tiny few individuals which are naturally resistant to certain antibiotics. This is because of the random nature of mutations.


When an antibiotic is applied, the initial innoculation will kill most bacteria, leaving behind only those few cells which happen to have the mutations necessary to resist the antibiotics. In subsequent generations, the resistant bacteria reproduce, forming a new colony where every member is resistant to the antibiotic. This is natural selection in action. The antibiotic is "selecting" for organisms which are resistant, and killing any that are not.​

I was about to post that myself!

Five Proofs of Evolution Evolution FAQ

 

[Uncensored2008]

So you admit that there is no factual evidence. I accept your surrender.

By "no factual evidence," you mean "overwhelming and irrefutable evidence," then?

Evolution is a fact - there is no debate.

 

[eots]

So you admit that there is no factual evidence. I accept your surrender.

By "no factual evidence," you mean "overwhelming and irrefutable evidence," then?

Evolution is a fact - there is no debate.

its all assumption..it could all be evidence of a common creator using the same source material

 

[Steven_R]

No, he is not wrong. And neither was Dr. Kent Hovind wrong which was proven when he defeated the evolution promoters at university after university. Perhaps that is why the Govt. trumped up charges against him and put him in prison for the past 99 months and counting - for depositing 9600 dollars into his own bank account. Utterly ridiculous. You people cannot debate the facts and win so you put those who present them best behind bars. What weakness!

I'm convinced at this point that you're parodying the Bible Bangers.

It's been a magnificent trolling effort. No joke, I got reeled in. Kudos to you.