Stroke-like brain damage found in coronavirus victims

irosie91

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The linked article in the OP states:

"Scientists have identified hallmarks of brain damage in a handful of patients after they died of coronavirus complications"

One would think that AFTER DEATH there would be "brain damage" after all the brain is....well....DEAD!! :auiqs.jpg:
after YOU die---your brain can be removed from you SKULL----and placed in formalin-----after several days it get firmed up and can be sliced up and examined----but by bit microscopically.
It is lots of fun. Got a citation---I am interested
in the kind of "damage" to which they are referring
 

irosie91

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On 2 Jul 2020, we posted on C-19 and autopsies:

2 Jul 2020 Post #667 Coronavirus Autopsies: A Story of 38 Brains, 87 Lungs and 42 Hearts
’....dengue....megakeryocytes’
Again, megakeryocytes are NOT virus.
As the name implies, they are "mega", they are extremely large cells.
Viruses are tiny.
They are not only small compared to normal cells, but even much more so compared to these huge megakeryocyte cells, which obviously are from the lungs.
So what they are actually trying to tell you, is that the person's own lung cells had been ripped apart from the immune system's out of proportion response.
It has nothing to do with a virus.
The virus is onl what trigger is.

And by the way, the vaccine might also trigger this response.
It has to also stimulate the immune system.
Megakaryocyte is a bone marrow thing---a big
irregular looking SPLOTCH----that makes platelets. It may have other functions---I am trying to remember
 

badger2

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The report in post #79, megakaryocytes were found in the COVID-19 hearts, though we’ll retain the image of neuro-dengue in spite of lack of megakaryocytes in COVID-19 brain autopsies. The reason is the GATA genes. We’ll first try to rescue the GATA connection to COVID-19.

Jun 1999 FOG 2 (Friend of GATA) / Brain / Megakaryocytes

Dec 2020 Dysregulated GATA-Binding Factor 3-Expressing T Cells / COVID-19

Earlier, what we found was that GATA 3 links dengue to t-bet, t-bet links COVID-19 and lupus, lupus links to chloroquine and chloroquine links back to the U.S. Army’s chloroquine-Bungarus study Chinese propaganda is now reifying in their media.

On 12 Jul 2020, we linked the GATA1 & 2 gene to mutations in megakaryocytes, GATA 3 to tonsillar cytokine expression, then the dengue link to t-bet, which links C-19 to lupus, lupus to chloroquine and completing the circle back to Bungarus for the U.S. Army study on another thread. There was only one case of C-19 linking tonsils in Germany:

12 Jul 2020 GATA 1, 2, & 3 / Megakaryocyte Mutations / Tonsillar Cytokine Expression, Posts # 736, 737, 740
 

badger2

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Yes, one can say that the host’s own immune response caused the brain damage, though not without help from the virus, which is doing other things behind its back. Next we’ll connect GATA, T-bet and IL-10 to dengue 2 in Taiwan:

Taiwan / GATA-3 mRNA / Dengue 2 / T-bet / IL-10

and IL-10 in COVID-19 is a disease severity predictor:

IL-6 & IL-10 / COVID-19
 

Rigby5

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But the point it is wrong to claim the covid-19 virus is doing organ damage.
That would imply the virus travels through the blood stream to all the organs, and it does NOT!
It can not, and instantly is killed and destroyed by contact with blood.

Some of those with covid-19 are experiencing a "cytokine storm" from an over active immune system response, and that is macerating the lungs with phages and other attempts to clean up what the immune system thinks are compromised tissue. And it is the debris from this attack by the immune system that is causing all the problems.
It is not the covid-19 virus itself.
And although it is the covid-19 that triggers it originally, we need to keep the distinction because we can't treat a virus, but we can treat an over active immune system.
 

Leo123

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after YOU die---your brain can be removed from you SKULL----and placed in formalin-----after several days it get firmed up and can be sliced up and examined----but by bit microscopically.
It is lots of fun. Got a citation---I am interested
in the kind of "damage" to which they are referring
They were talking out their ass basically.
 

Rigby5

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On 2 Jul 2020, we posted on C-19 and autopsies:

2 Jul 2020 Post #667 Coronavirus Autopsies: A Story of 38 Brains, 87 Lungs and 42 Hearts
’....dengue....megakeryocytes’
Again, megakeryocytes are NOT virus.
As the name implies, they are "mega", they are extremely large cells.
Viruses are tiny.
They are not only small compared to normal cells, but even much more so compared to these huge megakeryocyte cells, which obviously are from the lungs.
So what they are actually trying to tell you, is that the person's own lung cells had been ripped apart from the immune system's out of proportion response.
It has nothing to do with a virus.
The virus is onl what trigger is.

And by the way, the vaccine might also trigger this response.
It has to also stimulate the immune system.
Megakaryocyte is a bone marrow thing---a big
irregular looking SPLOTCH----that makes platelets. It may have other functions---I am trying to remember
Exactly.
Megakaryocyte has absolutely nothing at all to do with covid-19 viruses, other than it is covid-19 that triggers the cytokine storm that does all the damage.
Anyone claiming that finding traces of megakaryocyte in the brain shows that covid-19 got to the brain, is totally and completely wrong.
Covid-19 does not get into the blood at all, and most certainly can't get into the brain.
 

Rigby5

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The report in post #79, megakaryocytes were found in the COVID-19 hearts, though we’ll retain the image of neuro-dengue in spite of lack of megakaryocytes in COVID-19 brain autopsies. The reason is the GATA genes. We’ll first try to rescue the GATA connection to COVID-19.

Jun 1999 FOG 2 (Friend of GATA) / Brain / Megakaryocytes

Dec 2020 Dysregulated GATA-Binding Factor 3-Expressing T Cells / COVID-19

Earlier, what we found was that GATA 3 links dengue to t-bet, t-bet links COVID-19 and lupus, lupus links to chloroquine and chloroquine links back to the U.S. Army’s chloroquine-Bungarus study Chinese propaganda is now reifying in their media.

On 12 Jul 2020, we linked the GATA1 & 2 gene to mutations in megakaryocytes, GATA 3 to tonsillar cytokine expression, then the dengue link to t-bet, which links C-19 to lupus, lupus to chloroquine and completing the circle back to Bungarus for the U.S. Army study on another thread. There was only one case of C-19 linking tonsils in Germany:

12 Jul 2020 GATA 1, 2, & 3 / Megakaryocyte Mutations / Tonsillar Cytokine Expression, Posts # 736, 737, 740

You are not making a thesis statement to explain what it is your intent is?
If you are trying to figure out how the covid-19 triggers the cytokine storm, so that we can then detemine an immuno suppressant therapy to treat and prevent it, that would make sense.
But it would help if you were to say so.

If instead you are trying to show existing research that could have lead to covid-19 being deliberately enhanced, that is impossible to prove and does not really matter. These sorts of new novel hybrids are going to always happen anyway, and it does no good not to go back and point fingers.
 

badger2

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We look purely at the mechanisms that express the physics, what genes are triggered, how to connect the dots so as to build an image of C-19 biology.
 

justinacolmena

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Scientists have identified hallmarks of brain damage in a handful of patients after they died of coronavirus complications
There's a railroad and people have too much “family” -- and a COVID-19 diagnosis is a perfect cover-up for “the usual” murder-for-hire perpetrated by feuding families on unwanted relatives. The requisite “closure” for the family in lieu of the mental health diagnosis so often served on an involuntary basis.
Other street terms for being initiated into the Mafia include being "straightened out" or "baptized", and earning one's "badge".[1] "The books are open" is a phrase used in the Mafia to indicate that a particular Mafia family is ready to accept new members; in contrast, if a family is unwilling or unable to accept new members, the "books are closed." In Sicily, the proper term for a member of the Sicilian Mafia is in Italian uomo d'onore, or in Sicilian omu d'onuri. Mafioso is a common term used colloquially and by the press and academics, but it is generally not used by members of the Italian-American and Sicilian Mafia themselves.
 

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