Pfizer COVID-19 vaccine linked to rare blood disease - Israeli study

Science happens to be on OP’s side. Fau Chi knew that the virus would target elderly with compromised immune systems. They didn’t have to be infected with HIV-1.
 
This is just another dangerous effect of these vaccines... there are so many... heart, paralysis and the list goes on and on....it's so depressing what they are doing to the people....they are killing them.....a slow death....


This report from hemato-oncology does not go into detail about the disease, which includes susceptibility to cancer. For inherited thrombocytioipenia, there’s links to lipid rafts, target of SARS-CoV-2, so obesity should be considered because the vaccine may not know everything it is doing. Further, no genetic background of the patient is given, so the reader does not know to what degree this vaccine reaction is familial.
 
And at this point Hooper’s OPV theory of HIV/AIDS should not yet be dismissed. Due to cases of paralysis, elderly/immunodeficiency, etc. there is more investigating to do.
 
We doubt that Fau Chi has the stones to talk about this in public, though as coincidental as the same mutating position in a protein (an inherited thrombocytopenia gene, D485G compared with D485G in the mutant protein described below), what’s interesting is the D614G mutation Fau gets backwards in his video. We thought it odd when first hearing it, because a mutation to glycine is not only a mutation towards increased folding kinetics, it’s a very ancient amino acid, one of the two most abundant from the Miller-Urey volcanic spark experiments of 1959. There is something special Fau knows about this SARS-CoV-2 mutation. Thus, we investigate:

D485G / Trapping of Mutant Proteins

When and Where the Animal Model Matches the Patient
’....While PAH is assumed to develop in patients over months or years
, pulmonary hypertension in experimental animal models develops in days or weeks.’

We’ve already mentioned the odd COVID-19 autopsy occurrence of megakaryocytes in the heart, and there will be more to say about them, for their maturation is compromised in the mutated genes of inherited thrombocytopenia.

Interestingly, animal models may diverge at this point regarding civets as reservoirs of SARS-CoV:

Feb 2021 Kiev, The Ukraine; Georgetown University, Washington, D.C.
SARS-CoV-2 / PAH
’....Our histological evaluation revealed that patients who died of COVID-19 exhibited thickened pumonary vascular walls, one important hallmark of pulmonary arterial hypertension. By contrast, such pulmonary vascular remodeling lesions were not found in patients who died of SARS-CoV-1 during the 2002-2004 SARS outbreak or due to infection by H1N1 influenza.’
 
When was the Tongguan copper mine in Mojiang first opened and what would Edward Hooper make of these findings with regard to what he already knows about poliovirus an d HIV-1/HIV-2?
 
skye
The sciences disagrees with you

The science does not yet say if the mRNA vaccines are a good idea or not, because the immune system has memory, and the tampering we do now may last a life time.
And what these mRNA vaccines do is target a single spike protein, one our own exosomes also use.
That has very high long term risks.
 
The science does not yet say if the mRNA vaccines are a good idea or not, because the immune system has memory, and the tampering we do now may last a life time.
And what these mRNA vaccines do is target a single spike protein, one our own exosomes also use.
That has very high long term risks.
Here’s one telling aspect to it all: the 2021 Nobel prize winners- not a one from messenger RNA technology. Reportedly, one of the prize decision-makers made a statement something to the effect that they “wanted to make sure to reward technology that would be used in the future”. Indeed. Public statements to calm backers of mRNA- “bad timing with February as deadline for entries”. Hmm.
 
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A VAERS report links thromboembolism to thrombocytopenia and the first deaths of the Tongguan copper miners in Ap 2012, from which mine came SARS-CoV-2’s closest relative:

’....There was one patient who experienced a pulmonary thromboembolism and was reported to have thrombocytopenia; however, minimal case details were available.’

’....Case 1....There was a possibility of pulmonary thromboembolism. However, the patient was severely ill, it was not recommended to have an intensified chest CT check-up. Therefore, acute pulmonary infection could be one of the causes of death. The family of the patient refused the autopsy procedure.’
 
Post #52 mentioned fatal Heartland virus with thrombocytopenia in Tennessee. We’ll connect Heartland virus-caused amyloidosis to a COVID-19 patient, which secondary sclerosing cholangitis links to the Mengla filovirus in Yunnan, isolated from a proven Marburg filovirus reservoir genus, Roussetus, fruit bat. Both poliovirus and HIV-1 infections will figure into the investigation, and ebola filovirus vaccine links to SARS-CoV-2 Delta variant mutation, D614G.

Sep 2014 Fatal Heartland Virus
’....and a remote cerebral infarct and systemic amyloidosis.’

Secondary Sclerosing Cholangitis
’Table. Causes of Biliary Strictures....AIDS cholangiopathy....cholangiocarcinoma....amyloidosis.’

Rousettus fruit bat cancers include cholangiocarcinoma, and serious COVID-19 complications include requirement for liver transplant.

Ap 2021 Seattle: COVID-19 / Bile Duct / Secondary Sclerosing Cholangitis in a Critically-Ill Patient (SSC-CIP)
’....liver transplant....’

Edward Hooper’s OPV theory may have been prematurely dismissed. At any rate, AIDS cholangiopathy links to what Fau Chi, Baric, Zheng-li Shi and Daszak know about Afircan connections to Yunnan, Mengla filovirus source not being far away from Tongguan copper mine for RaTG13.

CD155 is the receptor for poliovirus.

Anhui Provincial Hospital, Hefei, China: CD155 / Cholangiocarcinoma
’....CD155 was positively associated with vascular endothelial growth factor and microvessel density in CCA tissues....poor prognosis....arises from bile duct epithelial cells.’
 
Bottlecap first made the Obama—Biden connection to funding of Duke-NUS Singapore on another USMB thread. This study directly relates to Duke-NUS and the cholangiocarcinoma-causing parasite, Opisthorchis viverrini:

Mar 2016 Australia / Duke-NUS Singapore / O. viverrini
’....The number of calcium ion channels classified for O. viverrini was higher than for C. elegans and humans.’

Ivermectin: 95.2% Cure Rate for O. viverrini

For those who wish to leave the “wormer” troglodytes behind, importin is the target and HIV-1 and dengue are the viruses.
Less than a month after the Mojiang copper miners entered the hospital, this would be big news for Fau Chi at NIH:

May 2012 Ivermectin HIV-1 / Dengue Inhibition
 
Thrombocytopenia (low platelet count) breaking through the vaccines is linked to ivermectin, because the site of platelet biogenesis is the lungs. Ivermectin has a long half-life and stays in the lungs. Homo sapiens’ most absurd mistake with the communist Chinese virus was to not have given ivermectin passing through a ventilator to a model animal such as the SARS-CoV-2-infected ferret at Kissimee. At this point, one of the suspect proteins in vaccine breakthrough linked to thrombocytopenia may be nucleolin, as the investigation continues in the search for mRNA links.
 
Nucleolin links to Fau Chi’s opposite number, Yong-zhen Zhang (Beijing CDC), who wrote on the fatal Huaiyangshan tick-borne virus and the fatal Heartland virus from the same tick, Haemaphysalis longicornis. Zhang writes during the same year, 2006, that Wuhan bat lady, Zheng-li Shi published on white spot syndrome of shrimp, noting that SARS-CoV-2 “Patient Zero” was supposedly a shrimp seller.

Nucleolin / Thrombocytopenia / E. Coli O157:H7 / HUS

2006 Yong-zhen Zhang, CDC Beijing / E. coli O157:H7

Zhang already links to Kunming Zoology Institute, so one may wish to know about Zhang’s cardiology if SARS-CoV-2 is indeed a thrombotic disease:

Jul 2003 Acute Coronary Syndrome, Dept. of Cardiology, Third Hospital of Peking University
 
Note that the stent in cardiology is linked to metal as is the copper miners’ pneumonia.
 
How is it possible, then, for SARS-CoV-2’s closest relative, RaTG13, to come from a copper mine? Free-floating spikes are reminiscent of the mRNA vaccines:

Nov 2015 Destruction of Coronaviruses on Copper Surfaces
’....Exposure to Cu destroyed the viral genomes and irreversibly affected virus morphology, including disintegration of envelope and dispersal of surface spikes....Cu(I) and Cu(II) moieties were responsible for the inactivation....animal coronaviruses that “host jump” to humans result in severe infections with high mortality.’

One of the contradictions linked to the Tongguan copper mine was the host-jumping idea, which was brought up by the Kunming Hospital staff, as per the Puna report and thesis. It questioned Zhengh-li Shi’s earlier proposal that these bat coronaviruses could not jump into humans. The bat source, Rhinolophus affinis, was confirmed by Yong-zhen Zhang’s Kunming Institute of Zoology.

Thus, the idea is that the virus was evolving into a copper-resistant coronavirus with increased affinity to lung ACE2 receptors, recalling that biogenesis of platelets begins in the lungs. RaTG13 sequence differences compared to SARS-Cov-2 may reflect this newly-acquired resistance and capacity, a difference of less than 4%.
 
The copper connection from Yong-zhen Zhang’s Beijing CDC is from 2005.

Jun 2005 Beijing CDC / Copper Inactivation of SARS-CoV
 
A knowledge gap exists in the monitoring of copper in platelets of athletes. First, Edward Hooper on 24 Ap 2020, states:

www.aidsorigins.com/covid-19-and-the-origins-of-aids-debate/#more-226
’I will, however, add one further detail that I believe has not yet been discussed in main stream media. The CISM Military World Games were held in Wuhan between 18-27 Oct 2019....attended by more than 9,000 athletes.’

Elsewhere, Hooper states that were reports of illness from this group.

Comparing exertions of bats for flight to human athletes, there is a copper depletion in human athlete erythrocytes while platelets also follow a similar trend:

2021 Spain: Copper Levels in Athletes
 

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