Ivermectin works for Covid and Per reviewed study from Peru proves it

[badger2]

Is ivermectin produced by Streptomyces?


Ivermectin is an antiparasitic from a family of chemicals called avermectins. These are macrocyclic lactones made from the fermentation broth of the fungus Streptomyces avermitilis. The macrolide ivermectin is available as an injectable, a spray, and an oral formulation.

--

wonder where some people get their info and how it is verifiable?

Get a grip Mary. Streptomyces produces avermectins. If you can read English you can verify that what we are saying is true. Streptomyces avermitilis endonuclease VII contains the furin-like sequence, RRYR.

Proof:
Streptomyces avermitilis MA-4680 (phiSAV)

Streptomyces avermitilis MA-4680 = NBRC 14893 DNA, complete genome - Nucleotide - NCBI

ncbi.nlm.nih.gov

'....putative endonuclease VII: RRYR.'

Most know by now that Michael Worobey is a close crony of The Elf, Anthony Fauci. Worobey's article on viruses of coelacanths link a gammaretrovirus, so we're not too concerned about what The Elf did during the HIV/AIDS era, or the posts about that already on this thread. We want to know why all the pretending not to notice that Streptomyces compounds were used successfully in Uttar Pradesh to prevent SARS2 infections.

Worobey's coelacanth virus links to Mozambique, where Wuhan Bat Lady, Zheng-li Shi, travelled to conference. Worobey's virus also has the same RRYR motif, so the origin of ivermectin links to the POS Elf and Michael Worobey's knowledge of the furin cleavage site, which was engineered into SARS2 sarbecovirus spike protein.

 

[badger2]

Worobey's gammaretrovirus (HIV = Retroviridae) is from the Mozambique Channel, Africa. Chinese communists made a mistake by allowing Zheng-li Shi to go to Mozambique, which lets the cat out of the bag for those of us who are clued up about Zoonati propaganda.

 

[Dante Reawakened]

Get a grip Mary. Streptomyces produces avermectins. If you can read English you can verify that what we are saying is true. Streptomyces avermitilis endonuclease VII contains the furin-like sequence, RRYR.

Proof:
Streptomyces avermitilis MA-4680 (phiSAV)

Streptomyces avermitilis MA-4680 = NBRC 14893 DNA, complete genome - Nucleotide - NCBI

ncbi.nlm.nih.gov

'....putative endonuclease VII: RRYR.'

Most know by now that Michael Worobey is a close crony of The Elf, Anthony Fauci. Worobey's article on viruses of coelacanths link a gammaretrovirus, so we're not too concerned about what The Elf did during the HIV/AIDS era, or the posts about that already on this thread. We want to know why all the pretending not to notice that Streptomyces compounds were used successfully in Uttar Pradesh to prevent SARS2 infections.

Worobey's coelacanth virus links to Mozambique, where Wuhan Bat Lady, Zheng-li Shi, travelled to conference. Worobey's virus also has the same RRYR motif, so the origin of ivermectin links to the POS Elf and Michael Worobey's knowledge of the furin cleavage site, which was engineered into SARS2 sarbecovirus spike protein.

You lose arguments and people when it becomes obvious your screeds are nothing but personal attacks on Faucci

 

[badger2]

You lose arguments and people when it becomes obvious your screeds are nothing but personal attacks on Faucci

You can't debate the science so rely on illusory ad hominems. The hominems are aptly placed, and you'd faila pop quiz for this thread.

 

[badger2]

So, the stupid in this thread can't and won't discuss the objective science we have posted. They are typical, unoriginal USMB POS.

So, Worobey's link to the coelacanth retrovirus (above) is a link to the Jesuit-trained piece of shit, Anthony Fauci for HIV-1. The Elf's dream was to create an AIDS vaccine, and RFK Jr. has aptly recorded Fau's troubling trajectory about this, a trajectory that includes astroturf covering over the caskets of children killed in Fauci's experiments in New York state.

We delve further into this deceptive psychopath from the NIH and his cronies by making necessary connections between SARS2, ivermectin, and HIV-1.

The Elf, Anthony Fauci, once shook hands with Big Farmer Gates on the shore of Lake Washington near Seattle.

SARS2 uses an endonuclease. It links to a study located in Seattle:

Aug 2023 U. Washington / SARS2 NSP15 Host Immune Evasion / Endonuclease

Regulation of coronavirus nsp15 cleavage specificity by RNA structure - PubMed

SARS-CoV-2, the etiologic agent of the COVID-19 pandemic, has had an enduring impact on global public health. However, SARS-CoV-2 is only one of multiple pathogenic human coronaviruses (CoVs) to have emerged since the turn of the century. CoVs encode for several nonstructural proteins (nsps)...

pubmed.ncbi.nlm.nih.gov

Above, we have already shown the furin-like site for the Streptomyces phage: endonuclease VII. S avermictilis, which produces ivermectin, is also infected with a virus (phage), which integrate into the genomes of that host.

(1985) S. avermictilis Phage Genome Integration

Isolation and preliminary characterization of lytic and lysogenic phages with wide host range within the streptomycetes - PubMed

Examination of approximately 700 soil samples yielded about 100 actinophages. Restriction analysis of phage DNA indicated that 57 are unique, and of these, 20 produce turbid plaques on one or more of the streptomycetes tested. Five phages are shown to insert into the genome of Streptomyces...

pubmed.ncbi.nlm.nih.gov

By 1985, The Elf's lab would have already known about phage integration in the producing organism of ivermectin.

HIV-1 Endoribonuclease Inhibition

Computational and Crystallographic Analysis of Binding Structures of Inhibitory Compounds for HIV-1 RNase H Activity - PubMed

Chemotherapy of human immunodeficiency virus type-1 (HIV-1) has significantly developed over the last three decades. The emergence of drug-resistant variants is, however, still a severe problem. The RNase H activity of HIV-1 reverse transcriptase is an attractive target for a new class of...

pubmed.ncbi.nlm.nih.gov

HIV-1 Pericytes / Endoribonuclease

Occludin Regulates HIV-1 Infection by Modulation of the Interferon Stimulated OAS Gene Family - PubMed

HIV-1-associated blood brain barrier (BBB) alterations and neurocognitive disorders are frequent clinical manifestations in HIV-1 infected patients. The BBB is formed by cells of the neurovascular unit (NVU) and sealed together by tight junction proteins, such as occludin (ocln). Pericytes are a...

pubmed.ncbi.nlm.nih.gov

Worobey's ancient fish gammaretrovirus links here:

Gammaretrovirus / Endoribonuclease / RNaseH Inhibition

Active site and allosteric inhibitors of the ribonuclease H activity of HIV reverse transcriptase - PubMed

Despite the wealth of information available for the reverse transcriptase (RT)-associated ribonuclease H (RNaseH) domain of lentiviruses, gammaretroviruses and long terminal repeat containing retrotransposons, exploiting this information in the form of an RNaseH inhibitor with high specificity...

pubmed.ncbi.nlm.nih.gov

Uneducated USMB POS will continue to look like clowns as they mouth off 'horse dewormer,' automatons that they are.

There is then, an avermectin link to endoribonucleases. By 1997, The Elf's labs knew why ivermectin is seldom selected against for resistance to the drug. That link is from Merck Research Labs in New Jersey:

(1997) Streptomyces RNase III is a Double-Strand-Specific Endoribonuclease

RNase III-Binding-mRNAs Revealed Novel Complementary Transcripts in Streptomyces - PubMed

<span><i>cis</i>-Antisense RNAs (asRNAs) provide very simple and effective gene expression control due to the perfect complementarity between regulated and regulatory transcripts. In <i>Streptomyces</i>, the antibiotic-producing clade, the antisense control system is not yet understood, although...

pubmed.ncbi.nlm.nih.gov

'The experiments presented in this paper predict that high level resistance to ivermectin may be rare because of the presence of several avermectin-sensitive GluCl channel subunits, each of which may need to be mutated to confer resistance.
....
Specific elimination of GluCl transcripts by RNase H digestion was employed to confirm that other C. elegans genes exist that can encode avermectin-sensitive GluCl channels.'

So again, as in this Merck report, ivermectin was first used against nematodes of mice, not worms of horses.


So yes, SARS2 Nsp 15 confers host immune evasion. It is a uridylate-specific endoribonuclease.

How Nsp 15 Changed Over the Course of the Pandemic

Biochemical Characterization of Emerging SARS-CoV-2 Nsp15 Endoribonuclease Variants - PubMed

Global sequencing efforts from the ongoing COVID-19 pandemic, caused by the novel coronavirus SARS-CoV-2, continue to provide insight into the evolution of the viral genome. Coronaviruses encode 16 nonstructural proteins, within the first two-thirds of their genome, that facilitate viral...

pubmed.ncbi.nlm.nih.gov

'....Like RNase A, Nsp 15 uses a two-step esterification mechanism to cleave RNA 3' of uridines....Nsp 15 mutations have been noted as clade-identifying markers.

Nsp 15 H235Y is a marker for Delta subclade C, K260R for clade E. Nsp 15 K13N identifies as a marker for subclade B.1.1.33 lineage in Brazil. Nsp 15 T113I is an Omicron marker, computationally predicted to have greatest impact fitness of Orf 1b (Nsp 12-16), across 6.4 million analyzed SARS2 genomes.'

 

[Delldude]

^^^ Idiotic statement that broke the internet today.

Oh really??/

 

[Deplorable Yankee]

Ivermectin Worked: New Peer-Reviewed Study Proves It - 🔔 The Liberty Daily

A preprint paper showing ivermectin’s effectiveness against COVID-19 in Peru convinced a group of doctors that widespread ivermectin distribution could end the pandemic in October 2020 Because the paper wasn’t yet peer-reviewed, it was brushed off; ivermectin for COVID-19 was vilified, as were...

thelibertydaily.com

Once again Fauci put profits over people like he did in the 80s with AZT. Wonder if the media will still call it horse paste?

Whole provinces in Peru, India, and other parts of the world used it and it worked ...safe ,cheap,and effective

Shut up and trust the Pfizer!

Africans eat ivermectin for everything and anything

Super safe cheap and effective

 

[billyerock123]

Thusfar it's your hearsay that's bullshit. Where are the citations for your conspiracy claim? RFK Jr. has citations for what he writes. This means that there are other people's work involved.

there's no hearsay here or it's my word ... its total factual information that Ivermectin does nothing for covid 19 ...RFK hasn't any citation that is credible... RFK has become the family idiot ... that they are all ashamed of ... His sources are fake ...his citations are fake ... everything about RFK jr. is fake ...get it moron stop using him as if he has value because he's a Kenedy ... he's using his name to sound important ... he's mentally deranged ... nobody in his entire family agrees with him at all on anything he says ...nobody in the democratic party accepts his citations as being credible ... nobody in the medical community accepts his citations ... stop playing with peoples lives with your bullshit post ... people are dying because of you

Ivermectin, an anti-parasitic drug used as the primary ingredient in canine heartworm preventatives, was touted as a “new tool” in the “arsenal” against SARS-CoV-2, the coronavirus strain responsible for the 2020 COVID-19 pandemic. Media publications like far-right cable channel OAN touted the drug as a “cure” that could kill the virus in “just 48 hours.”


Our analysis determined that this claim is a mixture of half-truths combined with misleading and overzealous claims.


Such claims began circulating online in April 2020 when Australian researchers with the Monash Biomedicine Discovery Institute and the Peter Doherty Institute of Infection and Immunity shared findings from an in vitro study that was in preprint and had not yet been peer reviewed by the scientific community. Similar claims made headway again in June, when the research was published in the scientific journal Antiviral Research, concluding that the FDA-approved drug ivermectin — which is most commonly used to treat internal and external animal parasites — was found to effectively stop SARS-CoV-2 virus from replicating in cells cultured in petri dishes. An in vitro study differs from an in vivo study in that the research occurs inside of extracted or grown cells cultured in a lab setting rather than in vivo, whereby the research takes place within a plant, human or animal. In vitro studies typically occur in the very early stages of research as a way to test pharmaceutical drugs or interventions before moving on to living organisms.


However, there is currently little evidence to suggest that ivermectin can “cure” COVID-19 in humans. That’s because throughout the course of the study, the drug was not given to people or animals, and the findings can only provide a potential path of future study into the safety and effectiveness of the drug.


In a separate study published in July 2020 in the Journal of Bangladesh College of Physicians and Surgeons, researchers described treating 100 patients in Bangladesh who tested positive for COVID-19 — an admittedly small sample size — with a combination treatment of ivermectin and doxycycline, along with “supportive treatment.” Nearly three-quarters of the patients were considered to have a “mild” case of the disease while one-in-ten were classified as “moderate,” which begs the question of whether the test subjects improved on their own accord or if it was some form of the various treatments given.


“It is too early in this pandemic to claim major successes of the effectivity of our combination therapy (Ivermectin and Doxycycline) as our number is small and there is no control group,” concluded the study authors. "Nevertheless, the results encourage us to continue the clinical study for people of the world in this improbable crisis as each and every life counts.”


There are studies underway to further determine whether ivermectin may be used in future human trials, though they have neither been peer-reviewed nor published in a reputable journal at the time of writing.


One such study is a U.S. clinical trial that took place between May 31 and July 27, 2020, in which researchers administered a preventative treatment of ivermectin to more than 200 asymptomatic individuals who had come in close contact with a confirmed COVID-19 patient but were not diagnosed with the disease themselves. Patients were measured for their development of COVID-19 symptoms within 14 days and diagnosis was then confirmed via a swab test. The preliminary and small-scale study, which has not been published in a peer-reviewed medical journal, suggested that ivermectin may be used to prevent infection of COVID-19. But again, the findings require further testing and evidence before they may be applied to larger human populations.


In March 2022, more than 3,500 patients diagnosed with symptomatic SARS-CoV-2 were analyzed in Brazil. Of those, 679 patients were randomly assigned to receive ivermectin, 679 a placebo, and the rest were given another medical intervention. Publishing their findings in the peer-reviewed scientific journal The New England Journal of Medicine, researchers concluded that there "were no significant effects of ivermectin use on secondary outcomes or adverse events."


"Treatment with ivermectin did not result in a lower incidence of medical admission to a hospital due to progression of Covid-19 or of prolonged emergency department observation among outpatients with an early diagnosis of Covid-19," wrote the study authors.


Ivermectin is a key ingredient in the canine heartworm preventative Heartguard. The anti-parasitic belongs to a class of medication called anthelmintics and is commonly given to animals via an oral tablet that works by either killing the parasites or stopping them from developing, according to the U.S. National Library of Medicine. Ivermectin is approved by the FDA for human use and is mainly prescribed in its topical form as a treatment for external parasites like head lice. In some cases, ivermectin may be prescribed as an oral tablet, a medication called Stromectol, to treat some parasitic roundworm infections.


Throughout the course of the pandemic, the scientific community rushed to find potential treatments for SARS-CoV-2, a single-stranded RNA virus closely related to SARS-CoV, the coronavirus responsible for the 2003 SARS pandemic. Scientists turned to previous SARS-related research in order to find treatments that may also be effective in treating COVID-19, one of which was ivermectin.


To test whether the anti-parasitic may have a similar effect on SARS-CoV-2, scientists cultured the cells of fetal pigs — again, not humans — in petri dishes in a laboratory setting before infecting the cultured cells with the coronavirus. When ivermectin was administered, it was shown to effectively stop SARS-CoV-2 replicating in these cultured cells. Within 24 hours of infection, there was a 93% reduction in viral replication and a 99.8% reduction by 48 hours. Though results are promising and show potential for the effective use of the drug as an antiviral in COVID-19 and other diseases, there is no clinical evidence to suggest that ivermectin can inhibit the replication of SARS-CoV-2 in humans. In short, it is far too early to claim that ivermectin is an effective cure or a treatment, or that it may become one in the near future.


In April 2020, the U.S. Food and Drug Administration (FDA) announced that such studies are common in the early stages of drug development and warned against self-medication until additional testing occurs. The FDA’s Center for Veterinary Medicine (CVM) issued a public letter following the April prepublication of the paper, expressing the agency’s concern about people who may self-medicate with ivermectin intended for animals. At the time of publication, there was no emergency use authorization for ivermectin in the U.S. to prevent or treat COVID-19, a legal measure that would bypass many regulatory laws.


“This type of study is commonly used in the early stages of drug development. Ivermectin was not given to people or animals in this study," wrote CVM Director Dr. Steven Solomon. "Additional testing is needed to determine whether ivermectin might be safe or effective to prevent or treat coronavirus or COVID-19.”


Solomon also said that the “FDA is concerned people should never take animal drugs, as the FDA has only evaluated their safety and effectiveness in the particular animal species for which they are labeled. These animal drugs can cause serious harm in people. People should not take any form of ivermectin unless it has been prescribed to them by a licensed health care provider and is obtained through a legitimate source.”


The ivermectin study prompted a comprehensive review published in The Journal of Antibiotics analyzing both in vitro and in vivo studies to determine the antiviral effects of ivermectin since 1970. Ivermectin was shown to have antiviral effects on Zika, dengue, yellow fever, and West Nile virus, among others — findings that further highlight that the anti-parasitic drug “could serve as a potential candidate in the treatment of a wide range of viruses including COVID-19.” Even so, the authors warned that clinical trials are still necessary to determine whether the drug is both safe and effective in human patients infected by SARS-CoV-2.


In short, the efficacy of ivermectin in combating COVID-19 depends on further preclinical testing and, if it were to pass, then the drug must undergo a four-part series of clinical trials as required by law. As of August 2020, it was too soon to draw conclusions about the effectiveness of ivermectin as a treatment or cure for COVID-19.

 

[billyerock123]

'Of course, the CDC never let on that it was foisting eight-month old data on Americans, allowing us to instead believe that these were current hospitalization rates as of August. To compound this flim-flam, CDC perpetuated an even more audacious hustle. CDC omitted the current (as of August) data related to hospitalizations from the Delta variant, which disproportionately hospitalized vaccinated individuals in those other countries for which we have more reliable data. CDC's promotion of this statistical bunko was obviously grossly misleading. Assuming President Biden wasn't deliberately lying fo the American people, it's clear that CDC was lying to President Biden and using him to dupe the rest of us.'
(RFJ Jr., ibid. p. 89)

So any more nazi dem exploitative flim-flam about the new variant, BA.2.86 will be noted, because like Delta, BA.2.86 is just as susceptible to ivermectin: it too has not mutated against ivermectin binding sites. This makes ivermectin on the cutting edge of world prophylaxis, in a replay of Uttar Pradesh.

the only one duped here is idiots who think like you ... who read conspiracy theorist like you do ... nothing you have said here has one bit of truth to it ...stop killing people with your stupidity ...

 

[IM2]

If the Biden administration had told you right wingers that ivermectin worked, you'd choose to take the vaccine.

 

[Burgermeister]

FDA never said doctors couldn't prescribe Ivermectin.

FDA couldn't give approval of Ivermectin or any other treatment as it would have killed the emergency use authorization and exposed big pharma to liability from usage of the Mrna cocktails.

Fifth Circuit sides with ivermectin-prescribing doctors in their quarrel with the FDA​

The Food and Drug Administration is not a physician, so it had no business cautioning people not to take ivermectin to ward off Covid-19 infections with social media posts stating, “You are not a horse,” the Fifth Circuit said.

Here is one of the items offered in proof of the FDA campaign against Ivermectin -



The article continues -
But the doctors say pharmacies stopped filling their prescriptions for the drug — which the FDA approved in 1996 for human use to treat parasitic diseases caused by round worms and black flies — after the FDA launched a public relations campaign in spring 2021 sounding the alarm that people were being hospitalized after self-medicating with large doses of the drug intended for deworming livestock that they had purchased over the counter.

This crap is so effective with the lemmings, especially the morons who use it to finally get a chance to feel what it's like to condescend (comically). They do it with masks and the jabs too.

 

[Faun]

So, the stupid in this thread can't and won't discuss the objective science we have posted. They are typical, unoriginal USMB POS.

So, Worobey's link to the coelacanth retrovirus (above) is a link to the Jesuit-trained piece of shit, Anthony Fauci for HIV-1. The Elf's dream was to create an AIDS vaccine, and RFK Jr. has aptly recorded Fau's troubling trajectory about this, a trajectory that includes astroturf covering over the caskets of children killed in Fauci's experiments in New York state.

We delve further into this deceptive psychopath from the NIH and his cronies by making necessary connections between SARS2, ivermectin, and HIV-1.

The Elf, Anthony Fauci, once shook hands with Big Farmer Gates on the shore of Lake Washington near Seattle.

SARS2 uses an endonuclease. It links to a study located in Seattle:

Aug 2023 U. Washington / SARS2 NSP15 Host Immune Evasion / Endonuclease

Regulation of coronavirus nsp15 cleavage specificity by RNA structure - PubMed

SARS-CoV-2, the etiologic agent of the COVID-19 pandemic, has had an enduring impact on global public health. However, SARS-CoV-2 is only one of multiple pathogenic human coronaviruses (CoVs) to have emerged since the turn of the century. CoVs encode for several nonstructural proteins (nsps)...

pubmed.ncbi.nlm.nih.gov

Above, we have already shown the furin-like site for the Streptomyces phage: endonuclease VII. S avermictilis, which produces ivermectin, is also infected with a virus (phage), which integrate into the genomes of that host.

(1985) S. avermictilis Phage Genome Integration

Isolation and preliminary characterization of lytic and lysogenic phages with wide host range within the streptomycetes - PubMed

Examination of approximately 700 soil samples yielded about 100 actinophages. Restriction analysis of phage DNA indicated that 57 are unique, and of these, 20 produce turbid plaques on one or more of the streptomycetes tested. Five phages are shown to insert into the genome of Streptomyces...

pubmed.ncbi.nlm.nih.gov

By 1985, The Elf's lab would have already known about phage integration in the producing organism of ivermectin.

HIV-1 Endoribonuclease Inhibition

Computational and Crystallographic Analysis of Binding Structures of Inhibitory Compounds for HIV-1 RNase H Activity - PubMed

Chemotherapy of human immunodeficiency virus type-1 (HIV-1) has significantly developed over the last three decades. The emergence of drug-resistant variants is, however, still a severe problem. The RNase H activity of HIV-1 reverse transcriptase is an attractive target for a new class of...

pubmed.ncbi.nlm.nih.gov

HIV-1 Pericytes / Endoribonuclease

Occludin Regulates HIV-1 Infection by Modulation of the Interferon Stimulated OAS Gene Family - PubMed

HIV-1-associated blood brain barrier (BBB) alterations and neurocognitive disorders are frequent clinical manifestations in HIV-1 infected patients. The BBB is formed by cells of the neurovascular unit (NVU) and sealed together by tight junction proteins, such as occludin (ocln). Pericytes are a...

pubmed.ncbi.nlm.nih.gov

Worobey's ancient fish gammaretrovirus links here:

Gammaretrovirus / Endoribonuclease / RNaseH Inhibition

Active site and allosteric inhibitors of the ribonuclease H activity of HIV reverse transcriptase - PubMed

Despite the wealth of information available for the reverse transcriptase (RT)-associated ribonuclease H (RNaseH) domain of lentiviruses, gammaretroviruses and long terminal repeat containing retrotransposons, exploiting this information in the form of an RNaseH inhibitor with high specificity...

pubmed.ncbi.nlm.nih.gov

Uneducated USMB POS will continue to look like clowns as they mouth off 'horse dewormer,' automatons that they are.

There is then, an avermectin link to endoribonucleases. By 1997, The Elf's labs knew why ivermectin is seldom selected against for resistance to the drug. That link is from Merck Research Labs in New Jersey:

(1997) Streptomyces RNase III is a Double-Strand-Specific Endoribonuclease

RNase III-Binding-mRNAs Revealed Novel Complementary Transcripts in Streptomyces - PubMed

<span><i>cis</i>-Antisense RNAs (asRNAs) provide very simple and effective gene expression control due to the perfect complementarity between regulated and regulatory transcripts. In <i>Streptomyces</i>, the antibiotic-producing clade, the antisense control system is not yet understood, although...

pubmed.ncbi.nlm.nih.gov

'The experiments presented in this paper predict that high level resistance to ivermectin may be rare because of the presence of several avermectin-sensitive GluCl channel subunits, each of which may need to be mutated to confer resistance.
....
Specific elimination of GluCl transcripts by RNase H digestion was employed to confirm that other C. elegans genes exist that can encode avermectin-sensitive GluCl channels.'

So again, as in this Merck report, ivermectin was first used against nematodes of mice, not worms of horses.


So yes, SARS2 Nsp 15 confers host immune evasion. It is a uridylate-specific endoribonuclease.

How Nsp 15 Changed Over the Course of the Pandemic

Biochemical Characterization of Emerging SARS-CoV-2 Nsp15 Endoribonuclease Variants - PubMed

Global sequencing efforts from the ongoing COVID-19 pandemic, caused by the novel coronavirus SARS-CoV-2, continue to provide insight into the evolution of the viral genome. Coronaviruses encode 16 nonstructural proteins, within the first two-thirds of their genome, that facilitate viral...

pubmed.ncbi.nlm.nih.gov

'....Like RNase A, Nsp 15 uses a two-step esterification mechanism to cleave RNA 3' of uridines....Nsp 15 mutations have been noted as clade-identifying markers.

Nsp 15 H235Y is a marker for Delta subclade C, K260R for clade E. Nsp 15 K13N identifies as a marker for subclade B.1.1.33 lineage in Brazil. Nsp 15 T113I is an Omicron marker, computationally predicted to have greatest impact fitness of Orf 1b (Nsp 12-16), across 6.4 million analyzed SARS2 genomes.'

Thanks for once again tacitly admitting a parasite is not a virus.

 

[badger2]

Thanks for once again tacitly admitting a parasite is not a virus.

No, a virus is a parasite of the cell. It hijacks the host's machinery (exploits it) and therefore is a parasite.

 

[badger2]

Before tending to post #188, it's noteworthy that McKernan mentioned being censored by an editor, so the politics is here, be sure of it.

McKernan Censored
Reinforcing what we've already posted about the producing organism of ivermectin, Streptomyces also being infected by a phage (virus), here is McKernan's now-defunct Twitter page as it first happened:

10 Ap 2023 Post # 2,121

COVID-19’s Biological Politics

Before the Metchnikoff excerpts, it is mandatory to link with current developments @Kevin_McKernan so as to make the sex-determining/transgender connections to furin and synchronize SARS2 origin investigations. Therefore, the pertinent article is this one, posted on 8 Ap 2023: '...."SARS2...

www.usmessageboard.com

'....SARS-CoV-2 awakens ancient retroviral genes and the expression of HERV-W....'

Phage infections are very likely to have prompted secondary metabolite synthesis of Streptomyces avermictilis avermectins.

 

[Burgermeister]

Thanks for once again tacitly admitting a parasite is not a virus.

Viruses are parasites.

 

[John Edgar Slow Horses]

Covid is a virus, not a parasite. A virus is a microscopic agent that infects living cells and replicates inside them, causing disease. A parasite is an organism that lives on or in another organism and benefits at the expense of the host. Covid does not fit the definition of a parasite because it does not have a cellular structure and it does not feed on the host's resources. Covid is a type of coronavirus, which is a family of viruses that cause respiratory infections in humans and animals. Bing AI search

 

[badger2]

In post #188, 2nd paragraph, the OAN URL does not function. Following that, the two institutions mentioned do not show the pertinent article from Monash. Scrolling down, the Antiviral Research article is the correct one, and mentions not pig cells, but Vero hSLAM cells (Vero [African Green Monkey Kidney]/ human signaling lymphocyte activation molecule) that were infected with SARS2 before adding the ivermectin.

RFK Jr. has published another article from Monash with a different title (The Real Anthony Fauci, page103): '"Lab Experiments Show Anti-Parasitic Drug, Ivermectin, Eliminates SARS-CoV-2 in Cells in 48 Hours." Monash Biomedicine Discovery Institute (3 Ap 2020)

https://www.monash.edu/discovery-institute/news-and-events/news/2020-articles/Lab-experiments-show-antiparasitic-drug-Ivermectin,-eliminates-SARS-CoV-2-in-cells-in-48-hours

E.C. Holmes was in Sydney, Australia at the time and would be one of the first to read the above Monash article, so there's already resonance with HIV/AIDS: Edward Hooper mentions E.C. Holmes on his webpage.

Edward Hooper / AIDS Origins
aidsorigins.com

Note the link between HIV-1 and African Green Monkey kidney cells, which Hooper mentions in his book, The River, which is now published online including the index.

Furthermore, E.C. Holmes is the second person on earth to see the published genome of SARS2, which was given to him by Beijing CDC's Yong-Zhen Zhang, who sequenced it.

 

[badger2]

This is correctly transcribed.

Lab experiments show anti-parasitic drug, Ivermectin, eliminates SARS-CoV-2 in cells in 48 hours

www.monash.edu

 

[badger2]

As the reader can see in the article above, Dr. Leon Caly (Royal Melbourne Hospital) never mentions E.C. Holmes but does say: " As the virologist who was part of the team who were first to isolate and share SARS-CoV-2 outside of China in January 2020, I am excited about the prospect of ivermectin being used as a potential drug against COVID-19."

 

[badger2]

Covid is a virus, not a parasite. A virus is a microscopic agent that infects living cells and replicates inside them, causing disease. A parasite is an organism that lives on or in another organism and benefits at the expense of the host. Covid does not fit the definition of a parasite because it does not have a cellular structure and it does not feed on the host's resources. Covid is a type of coronavirus, which is a family of viruses that cause respiratory infections in humans and animals. Bing AI search

No, COVID does indeed feed (and replicate) via the host's resources.