One can see Fig. 4A in the above report, "Expression Profiling of Computational Biomarkers," that Guttmacher's HOX genes are there linking anti-SARS-CoV-2 activity via the abortifacient Withania, chloroquine, and quaternary ammonia compounds: Fig. 4a: 'Activation of HOX genes during differentiation.'
Next, we link Guttmacher syndrome to Adams-Oliver syndrome (AOS)/Poland sequence and A.E. Guttmacher
Possible common pathogenetic mechanisms for Poland sequence and Adams-Oliver syndrome: an additional clinical observation
pubmed.ncbi.nlm.nih.gov
So linking AOS is this Turkish report for hand-foot-genital syndrome from 2008, which links the HOX genes. Notice that the mother does not know the name of the antibiotic that may have triggered the genetic anomalies, though it does resonate with scalp problems such as scabies (of which Paguma scabies was treated with ivermectin):
2008 Turkey / AOS / Aplasia Cutis Congenita of the Scalp, Skull, Dura Associated with Adams-Oliver
A 1-day-old boy with the characteristics of Adams-Oliver syndrome was presented. Adams-Oliver syndrome has a wide spectrum of anomalies ranging from aplasia cutis congenita, cutis marmorata telangiectatica congenita and transverse limb defects to lethal anomalies. Our patient had aplasia cutis...
pubmed.ncbi.nlm.nih.gov
'....the mother was treated with an antibiotic which she did not recognize the name for gastroenteritis during her pregnancy....vascular disruption....abnormal small vessels causing suspension of blood flow appearing in embryogenesis.'
We have already USMB posted gastrointestinal symptoms at Wuhan previous to the outbreak. That Harvard study is located in the USMB "COVID-19's Biological Politics" thread, post #892, 3 Dec 2021. In the Turkish report above, one can see in the photo that the brain is protruding from the skull of the AOS infant. The vascular coagulation symptom of the infant is similar in SARS-CoV-2 infection.
With HOX gene-induced hand-foot-genital syndrome, fertility is not affected:
en.wikipedia.org
'....fertility is normal....60% of mutations are polyalanine expansions.'