I have posted the reasons did you read them ?
Look back they went into detail by the amount of mutations that happen naturally not induced. And by the results we see that comes from mutations. Even scientist on your side know these arguments are a problem for the theory.
Look back through the thread they go into detail.
When we induced mutations in the drosophila most were harmful and all flies became weaker and died off prematurely. We also have observed many mutations that happened naturally. There was not much difference from the mutations that were induced and the ones that happened naturally.
By there being many more harmful and neutral mutations it is overwhelming in number compared to beneficial mutations. But even the beneficial mutations come at a price. How is a mutation a benefit to me if i have to have a blood disorder to fight off a disease ?
Yes, most mutations are harmful in those experiments. Yet similar experiments with yeast show a
7% rate of harmful mutations. You're are correct with the statement of neutral (that is, mutations that have little-to-no effect on an organism) and harmful mutations happening more often than beneficial ones. An important question to ask though, is what simply is the
rate of these mutations happening at? I can guarantee you these mutations don't happen nearly as often as you suggest by the fruit fly experiment, especially in other organisms. This is considering that these mutations are induced artificially via radiation which bumps up the mutation rate, and mutations don't actually happen like that in nature.
Another point is that organisms do in fact have DNA repair, repairing the majority of these mutations before they can actually have an effect. So every single mutation isn't simply allowed to occur.
I don't get your last question. Is the blood disorder caused by the beneficial mutation? Because your question sounds like it's pre-existing and unrelated.
No,I believe i was speaking of sickle cell.
3 - THE ONE "BENEFICIAL" MUTATION
SICKLE-CELL ANEMIA—Evolutionists point to sickle-cell anemia as the outstanding example of beneficial evolutionary change through mutation.
A long time ago, a mutation occurred in someone in Africa. As do all mutational changes, this one resulted in damage. In this instance, the shape of the red blood cells was changed, from its normal flattened shape, to a quarter-moon shape. Because it tended to cause serious anemia, instead of killing outright, sickle-cell anemia passed into the race and became a recessive factor.
The problem was that, although the blood of a person with sickle-cell anemia does not properly absorb food and oxygen,—that person, oddly enough, will be less likely to acquire malaria from the bite of an anopheles mosquito. As a result, the sickle-cell anemia factor has become widespread in Africa. This is the best example of a "beneficial" mutation that evolutionary scientists are able to offer us.
"Actually, only three evolutionists have ever given me an example of a beneficial mutation. It was the same example all three times: sickle-cell anemia . . Sickle-cell anemia is often given as an example of a favorable mutation, because people carrying sickle-cell hemoglobin in their red blood cells are resistant to malaria. But the price for this protection is high: 25 percent of the children of carriers will probably die of the anemia, and another 25 percent are subject to malaria.
"The gene will automatically be selected when the death rate from malaria is high, but evolutionists themselves admit that the short time advantages produce ‘mischievous results’ detrimental to long-term survival."—Henry Morris and Gary Parker, What is Creation Science? (1987), pp. 103, 104.
Actual statistics reveal that the death rate from malaria for normal people in certain parts of Africa is over 30 percent while only 25 percent of carriers of sickle-cell anemia are likely to contract it. But in return for the advantage, 25 percent of their children will die of this serious anemia.
These carriers have a 50-50 proportion of regular and sickle-cell red blood cells, but 25 percent of their children will have 100 percent sickle-cell RBCs, and will die as a result. The other 75 percent will also be carriers and have the 50-50 proportion of cells.
In sickle-cell anemia, one amino acid in a peptide of nine in a string is faulty. Valine is there instead of glutamic acid. That one change makes all the difference, changing regular hemoglobin into sickle-cell hemoglobin.
This outstanding example of a "beneficial mutant" not only damages those who have it, but in the process would normally eradicate itself. It is only the deaths caused by malaria that favor it.
"In regions where malaria is not an acute problem, the gene does tend to die out. In America, the incidence of sickle-cell genes among blacks may have started as high as 25 percent. Even allowing for a reduction to an estimated 15 percent by admixture with non-black individuals, the present incidence of only 9 percent shows that the gene is dwindling away. In all probability it will continue to do so. If Africa is freed of malaria, the gene will presumably dwindle there, too."—*Asimov’s New Guide to Science (1984), p. 619.
Mutations