Yes, here are the authors' Twitter pages:
PD-1 is current on Bruttel's page, so we'll link osteopontin to PD-1 and to Brook Jackson's troponin. If there is a legitimate amyloid phenotype to the vaccine-induced clotting (apparently accompanied by feed-forward cytokines), then we are free to speculate on this osteopontin-troponin assemblage:
Sep 2014 Heidelberg, Germany / Troponin / Osteopontin / Systemic Light-Chain Amyloidosis
These data demonstrated that OPN appears to be a valuable marker in the clinical routine for evaluation of patients with AL amyloidosis, especially if it is used in combination with cTNT and/or NT-proBNP.
pubmed.ncbi.nlm.nih.gov
There are 22 more Pubmed entries for this osteopontin-troponin assemblage.
Other genes implicated construct the investigative trajectory
Sep 2009 Japan / PD-1 / Osteopontin
Although altered T cell function plays a part in immunosenescence, the mechanisms remain uncertain. Here we identify a bona fide age-dependent PD-1(+) memory phenotype (MP) CD4(+) T cell subpopulation that hardly proliferates in response to T cell receptor (TCR) stimulation and produces abundant...
pubmed.ncbi.nlm.nih.gov
'....Here we identify a bona fide age-dependent PD-1(+) memory phenotype (MP) CD4(+) T cell subpopulation that hardly proliferates in response to T cell receptor (TCR) stimulation and produces abundant osteopontin at the cost of typical T cell lymphokines. These T cells demonstrate impaired repopulation in Rag2 (_?_) mice, but a homeostatic proliferation in gamma-ray-irradiated mice.
....
reval a unique molecular signature including strong expression of C/EBPalpha normally expressed i myeloid lineage cells....caused a striking activation of Spp1 encoding osteopontin along with concomitant repression of T cell lymphokine genes.'
The chron for these studies links (Gates-funded) Johns Hopkins and MIT during 2014, recalling elevated troponin expressed in either SARS2 or its mRNA "vaccine.":
Osteopontin / HIV-1
Osteopontin (OPN) is a proinflammatory cytokine produced by T-cells, macrophages, osteoclasts, and several other cell types, which confers immunity to many intracellular pathogens. OPN was first identified as an early marker of cellular activation of T-lymphocytes and subsequently was shown to...
pubmed.ncbi.nlm.nih.gov
'....OPN levels are elevated in the plasma and cerebrospinal fluid of HIV-infected individuals even more so in those suffering from HIV-related neurocognitive impairment....OPN-regulated cellular pathways are commandeered by HIV to promote productive replication and cell-to-cell spread.'
It thus becomes mandatory to introduce the RGD motif once again:
Dec 2020 Rome / HIV-1 / RGD Motif
Previous work has shown that the Tat protein of Human Immunodeficiency Virus (HIV)-1 is released by acutely infected cells in a biologically active form and enters dendritic cells upon the binding of its arginine-glycine-aspartic acid (RGD) domain to the α5β1, αvβ3, and αvβ5 integrins. The...
pubmed.ncbi.nlm.nih.gov
'....enters dendritic cells upon binding of its RGD domain to the alpha5beta1, alpha-v-beta3, alpha-v-beta5 integrins.'