That you would actually believe that the bible is a science book is not surprising but does show the breadth of your stupidity.
https://www.youtube.com/watch?feature=player_detailpage&v=h5ACyiSPAmE
I never said I thought the bible was a science book. I do believe there are things mentioned in the bible that have been confirmed by modern day science and technology. I guess you're getting angry over my questions.
Angry? Not in the least. You said "I would put my bible first before any secular science text book". That certainly does imply that you are using the bible to support your notions of science. I think it is disingenuous of you to make claims like the one you made that apes and humans don't share genetic defects, and when shown that you are wrong you don't acknowledge your error. I don't think you are being honest in your arguments, nor do I think you are learning anything from these discussions. So what's the point in having them in the first place?
Look, I am talking rare genetic disorders,animals suffer from cancer,heart disease. The apes do not suffer from Aids or Malaria.
nherited mutations of oncogenes
A few cancer syndromes are caused by inherited mutations of proto-oncogenes that cause the oncogene to be turned on (activated). For example, multiple endocrine neoplasia type 2 (MEN2) is caused by an inherited mutation in the gene called RET. People affected by this syndrome often develop an uncommon thyroid cancer called medullary cancer of the thyroid. They also develop other tumors, including pheochromocytoma and nerve tumors. Inherited mutations in the gene called KIT can cause hereditary gastrointestinal stromal tumors (GISTs). And inherited mutations in the gene called MET can cause hereditary papillary renal cancer.
Acquired mutations of oncogenes
Most cancer-causing mutations involving oncogenes are acquired, not inherited. They generally activate oncogenes by chromosome rearrangements, gene duplication, or mutation. For example, a chromosome rearrangement can lead to formation of the gene called BCR-ABL, which leads to chronic myeloid leukemia (CML). Acquired mutations that activate the KIT gene cause most cases of gastrointestinal stromal tumor (GIST).
Inherited abnormalities of tumor suppressor genes have been found in some family cancer syndromes. They cause certain types of cancer to run in families. For example, a defective APC gene causes familial adenomatous polyposis (FAP), a condition in which people develop hundreds or even thousands of colon polyps. Often, at least one of the polyps becomes cancer, leading to colon cancer. There are many examples of inherited tumor suppressor gene mutations, and more are being discovered each year. For more information about inherited mutations and cancer, see our document Heredity and Cancer.
Acquired mutations of tumor suppressor genes
Tumor suppressor gene mutations have been found in many cancers. Most of these mutations are acquired, not inherited.
For example, abnormalities of the TP53 gene (which codes for the p53 protein) have been found in more than half of human cancers. Acquired mutations of this gene appear in a wide range of cancers, including lung, colorectal, and breast cancer. The p53 protein is involved in the pathway to apoptosis. This pathway is turned on when a cell has DNA damage that can't be repaired. If the gene for p53 is not working properly, cells with damaged DNA continue to grow and divide. Over time this can lead to cancer.
Acquired changes in many other tumor suppressor genes also contribute to the development of sporadic (not inherited) cancers.
Gene mutations that can lead to cancer