In the following excerpt, Australia's Sharri Markson mentions UTMB Galveston (USMB search UTMB Galveston).
'Dr. Ebright says there are two other risky scientific experiments at the Wuhan Institute of Virology that are possible gain-of-function research and are of concern. One is a project called, "Two Mutations were Critical for Bat-to-Human Transmission of Middle East Respiratory Syndrome Coronavirus," published in June 205. It was done by Shi Zhengli, Baric and two scientists, Lanyiang Du and Shibo Jang from the Lindsley Kimball Research Institute's New York Blood Centre. The University of Minnesota Medical School's Fang Li, Chang Liu and Yang Yang were also co-authors.
The second is a research paper published in Aug 2020 titled, " A Zika Virus Envelope Mutation Preceding the 2015 Epidemic Enhances Virulence and Fitness for Transmission." This was done by the University of Texas Medical Branch, Galveston, in conjunction with the Wuhan Institute of Virology.
Because of the type of risky research underway at the Wuhan Institute of Virology, when SARS-CoV-2 virus broke out, Dr. Ebright instantly suspected it may have originated from the Institute....As Ebright has been at pains to point out, the type of gain-of-function research that has been undertaken worldwide in the last half-decade, including at the Wuhan Institute of Virology, does not leave signatures. You cannot tell from studying the virus if it has been subjected to genetic manipulation.'
(Markson, What Really Happened in Wuhan, p. 208)
The first mentioned article is here, and we will note the retroviruses that were pseudotyped as well as the arginines in the mutation 'RSVR':
To understand how Middle East respiratory syndrome coronavirus (MERS-CoV) transmitted from bats to humans, we compared the virus surface spikes of MERS-CoV and a related bat coronavirus, HKU4. Although HKU4 spike cannot mediate viral entry into human cells, two mutations enabled it to do so by...
pubmed.ncbi.nlm.nih.gov
'....To confirm that S746R mutation restored the hPPC motif, we produced retroviral pseudotyped with HKU4 spike in human cells....the hPPC and hEPC motifs being well documented as cleavage sites for human protease.'
Readers can scroll down to Fig. 1 to see the "critical arginine" represented in the MERS spike. Thus, the RRYR motifs in 1.) bcl-2 breast/lung cancer protein, 2.) the phage that infects Streptomyces avermitilis (producer of ivermectin) and 3.) the Coelacanth retrovirus, all link to this MERS example mentioned in Markson's book.
