What color shoes do you have? That question is just as relevant to this topic as yours.
.
BS - it's relevant as hell. People are sick of you america-hating liberals who won't put your money where your mouth is.
Whether I take in immigrant children is totally irrelevant to whether this virus is caused by immigrant children. But you know that, you just can't prove the latter so you are trying to change the debate. Tough. Your idiotic topic was just debunked, now get on with your life.
Thanks for proving two things:
1. You can't see the forest for the trees.
2. You are a hypocrite.
Please provide evidence that this virus is a result of the immigrant children. Otherwise I accept your pathetic attempt to change the topic as a concession.
You are completely missing the point. The U.S. has wiped out diseases that are chronic and deadly in other ares of the world due to lack of hygiene and vaccinations. With this open border policy, we are reintroducing them, and allowing new ones to enter.
There have been outbreaks of Chagas in California, for example, something that is hardly a native disease.
Chagas disease - Wikipedia the free encyclopedia
Chagas disease is a disease caused by an insect bite and the article you posted even says there is no vaccine for it.
In Chagas-endemic areas, the main mode of transmission is through an insect
vector called a
triatomine bug.
[4] A triatomine becomes infected with
T. cruzi by feeding on the blood of an infected person or animal. During the day, triatomines hide in crevices in the walls and roofs. The bugs emerge at night, when the inhabitants are sleeping. Because they tend to feed on people's faces, triatomine bugs are also known as "kissing bugs". After they bite and ingest blood, they defecate on the person. Triatomines pass
T. cruzi parasites (called
trypomastigotes) in feces left near the site of the bite wound.
[4]
Scratching the site of the bite causes the trypomastigotes to enter the host through the wound, or through intact
mucous membranes, such as the
conjunctiva. Once inside the host, the trypomastigotes invade cells, where they differentiate into intracellular
amastigotes. The amastigotes multiply by
binary fission and differentiate into trypomastigotes, which are then released into the bloodstream. This cycle is repeated in each newly infected cell. Replication resumes only when the parasites enter another cell or are ingested by another vector.
[4] (See also:
Life cycle and transmission of T. cruzi)
Dense vegetation (such as that of tropical
rainforests) and urban habitats are not ideal for the establishment of the human transmission cycle. However, in regions where the
sylvatic habitat and its fauna are thinned by economic exploitation and human habitation, such as in newly
deforested areas,
piassava palm culture areas, and some parts of the
Amazon region, a human transmission cycle may develop as the insects search for new food sources.
[12]
T. cruzi can also be transmitted through
blood transfusions. With the exception of blood derivatives (such as fractionated
antibodies), all blood components are infective. The parasite remains viable at 4 °C for at least 18 days or up to 250 days when kept at room temperature. It is unclear whether
T. cruzi can be transmitted through frozen-thawed blood components.
[13]
Other modes of transmission include
organ transplantation, through
breast milk,
[14] and by accidental laboratory exposure. Chagas disease can also be spread congenitally (from a pregnant woman to her baby) through the
placenta, and accounts for approximately 13% of stillborn deaths in parts of Brazil.
[15]
Oral transmission is an unusual route of infection, but has been described. In 1991, farm workers in the state of
Paraíba, Brazil, were infected by eating contaminated food; transmission has also occurred via contaminated
açaí palm fruit juice and
garapa.
[16][17][18][19][20] A 2007 outbreak in 103 Venezuelan school children was attributed to contaminated
guava juice.
[21]
Chagas disease is a growing problem in Europe, because the majority of cases with chronic infection are asymptomatic and because of migration from Latin America.
[22]
Diagnosis[edit]
The presence of
T. cruzi is diagnostic of Chagas disease. It can be detected by
microscopic examination of fresh anti
coagulated blood, or its
buffy coat, for motile parasites; or by preparation of thin and thick blood smears stained with
Giemsa, for direct visualization of
parasites. Microscopically,
T. cruzi can be confused with
Trypanosoma rangeli, which is not known to be
pathogenic in humans. Isolation of
T. cruzi can occur by inoculation into
mice, by culture in specialized media (for example, NNN, LIT); and by
xenodiagnosis,
[23] where uninfected
Reduviidae bugs are fed on the patient's blood, and their gut contents examined for parasites.
[10]
Various
immunoassays for
T. cruzi are available and can be used to distinguish among
strains (zymodemes of
T.cruzi with divergent pathogenicities). These tests include: detecting
complement fixation, indirect
hemagglutination, indirect
fluorescence assays,
radioimmunoassays, and
ELISA. Alternatively, diagnosis and strain identification can be made using
polymerase chain reaction (PCR).
[10]
Prevention[edit]

Awareness and prevention campaign poster in
Cayenne, French Guiana, 2008
There is currently no vaccine against Chagas disease
[24] and prevention is generally focused on fighting the
vector Triatoma by using sprays and paints containing
insecticides (synthetic
pyrethroids), and improving housing and sanitary conditions in rural areas.
[25] For urban dwellers, spending vacations and
camping out in the wilderness or sleeping at hostels or mud houses in endemic areas can be dangerous; a
mosquito net is recommended. Some measures of vector control include:
- A yeast trap can be used for monitoring infestations of certain species of triatomine bugs (Triatoma sordida, Triatoma brasiliensis, Triatoma pseudomaculata, and Panstrongylus megistus).[26]
- Promising results were gained with the treatment of vector habitats with the fungus Beauveria bassiana.[27]
- Targeting the symbionts of Triatominae through paratransgenesis can be done.[28]
A number of potential vaccines are currently being tested. Vaccination with
Trypanosoma rangeli has produced positive results in animal models.
[29] More recently, the potential of DNA vaccines for
immunotherapy of acute and chronic Chagas disease is being tested by several research groups.
[30]
Blood transfusion was formerly the second-most common mode of transmission for Chagas disease, but the development and implementation of
blood bank screening tests has dramatically reduced this risk in the last decade.
Blood donations in all endemic Latin American countries undergo Chagas screening, and testing is expanding in countries, such as France, Spain and the United States, that have significant or growing populations of immigrants from endemic areas.
[31][32] In Spain, donors are evaluated with a questionnaire to identify individuals at risk of Chagas exposure for screening tests.
[32]
The US FDA has approved two Chagas tests, including one approved in April 2010, and has published guidelines that recommend testing of all donated blood and tissue products.
[32][33] While these tests are not required in US, an estimated 75–90% of the blood supply is currently tested for Chagas, including all units collected by the
American Red Cross, which accounts for 40% of the U.S. blood supply.
[33][34] The Chagas Biovigilance Network reports current incidents of Chagas-positive blood products in the United States, as reported by labs using the screening test approved by the FDA in 2007.
[35]